Cialis works by increasing blood flow to the penis. This effect stems from its inhibition of phosphodiesterase type 5 (PDE5), an enzyme that breaks down cyclic guanosine monophosphate (cGMP). Higher cGMP levels relax the smooth muscles in the penis, allowing increased blood flow.
Specifically, the inhibition of PDE5 leads to increased nitric oxide (NO) activity. NO plays a crucial role in the erectile process by triggering a cascade of events that ultimately result in penile engorgement. Cialis enhances this natural process.
This mechanism differs slightly from other erectile dysfunction medications. While similar medications also target PDE5, Cialis boasts a longer half-life, resulting in a more sustained effect. This extended duration allows for flexibility in timing.
It’s important to consult a doctor before using Cialis. They can assess your overall health and determine if Cialis is appropriate for you, considering any potential interactions with other medications you may be taking.
